人 MTFR1L 抗体对
01AB00315
Mitochondrial fission regulator 1 like (MTFR1L) is a protein that plays a crucial role in regulating mitochondrial morphology and dynamics. It is localized to the outer mitochondrial membrane and is involved in the adaptation of mitochondrial dynamics to metabolic changes. MTFR1L is phosphorylated by AMP-activated protein kinase (AMPK), which controls its function in regulating mitochondrial morphology, including mitochondrial fission and fusion events. Loss of MTFR1L leads to mitochondrial elongation and increased mitochondrial fusion events, suggesting its importance in maintaining mitochondrial homeostasis. While there is no direct association with specific diseases, alterations in MTFR1L expression have been observed in response to various chemical exposures, such as bisphenol A and carbon nanotubes, which may have implications for cellular health.
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技术参数
- 靶点基因名
- MTFR1L
- 基因别名
- FAM54B
- 蛋白别名
- mitochondrial fission regulator 1 like; MTFR1L
- UniProt
- Q9H019
- 物种
- Human
产品详情
- 靶点名称
- MTFR1L
- 蛋白名称
- mitochondrial fission regulator 1 like
- 蛋白别名
- Mitochondrial fission regulator 1-like
- 靶点基因名
- MTFR1L
- 基因别名
- FAM54B
- UniProt
- Q9H019
- 来源物种
- Homo sapiens (Human)
- 物种反应性
- Human
背景介绍
Mitochondrial fission regulator 1 like (MTFR1L) is a protein that plays a crucial role in regulating mitochondrial morphology and dynamics. It is localized to the outer mitochondrial membrane and is involved in the adaptation of mitochondrial dynamics to metabolic changes. MTFR1L is phosphorylated by AMP-activated protein kinase (AMPK), which controls its function in regulating mitochondrial morphology, including mitochondrial fission and fusion events. Loss of MTFR1L leads to mitochondrial elongation and increased mitochondrial fusion events, suggesting its importance in maintaining mitochondrial homeostasis. While there is no direct association with specific diseases, alterations in MTFR1L expression have been observed in response to various chemical exposures, such as bisphenol A and carbon nanotubes, which may have implications for cellular health.