抗人 FAM13C 抗体对
01AB03869
Protein FAM13C (FAM13C) is a protein implicated in various cellular processes, including cell signaling and proliferation. It is characterized by the presence of a coiled-coil domain, which is thought to facilitate protein-protein interactions. FAM13C has been associated with the regulation of the RhoA signaling pathway, influencing cytoskeletal dynamics and cellular migration. Dysregulation of FAM13C has been linked to several diseases, particularly cancer. For instance, studies have shown that altered expression of FAM13C can contribute to tumorigenesis in lung and breast cancers, where it may act as a tumor suppressor. In lung cancer, low levels of FAM13C are correlated with poor prognosis and aggressive tumor behavior. Additionally, FAM13C has been implicated in the development of chronic obstructive pulmonary disease (COPD), where its expression may influence airway remodeling and inflammation.
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技术参数
- 靶点基因名
- FAM13C
- 基因别名
- FAM13C1
- 蛋白别名
- family with sequence similarity 13 member C; FAM13C
- UniProt
- Q8NE31
- 物种
- Human
产品详情
- 靶点名称
- FAM13C
- 蛋白名称
- family with sequence similarity 13 member C
- 蛋白别名
- Protein FAM13C
- 靶点基因名
- FAM13C
- 基因别名
- FAM13C1
- UniProt
- Q8NE31
- 来源物种
- Homo sapiens (Human)
- 物种反应性
- Human
背景介绍
Protein FAM13C (FAM13C) is a protein implicated in various cellular processes, including cell signaling and proliferation. It is characterized by the presence of a coiled-coil domain, which is thought to facilitate protein-protein interactions. FAM13C has been associated with the regulation of the RhoA signaling pathway, influencing cytoskeletal dynamics and cellular migration. Dysregulation of FAM13C has been linked to several diseases, particularly cancer. For instance, studies have shown that altered expression of FAM13C can contribute to tumorigenesis in lung and breast cancers, where it may act as a tumor suppressor. In lung cancer, low levels of FAM13C are correlated with poor prognosis and aggressive tumor behavior. Additionally, FAM13C has been implicated in the development of chronic obstructive pulmonary disease (COPD), where its expression may influence airway remodeling and inflammation.